bims-simsho Biomed News
on Systems immunology and sex hormones
Issue of 2025–06–29
sixteen papers selected by
Chun-Chi Chang, Lunds universitet



  1. Acta Biochim Pol. 2025 ;72 14241
       Background: The postpartum period involves complex physiological changes, notably in hormone levels, that significantly influence immune system function. Hormonal regulation during pregnancy prevents maternal immune rejection of the fetus, but following childbirth, these hormone levels drop rapidly, leading to immune reconstitution.
    Aim: This review investigates the impact of hormonal changes on immune system dynamics during the postpartum period and highlights their implications for maternal recovery.
    Methods: The study analyzed current literature, focusing on hormonal influences, particularly cortisol, prolactin, estrogen, and progesterone, on immune reconstitution with associated inflammatory responses in the postpartum period.
    Results: Postpartum immune reactivation, triggered by hormonal shifts, can lead to a resurgence of inflammatory reactions. This process, characterized by increased cortisol and prolactin levels and a rapid decline in estrogen and progesterone, could exacerbate dormant autoimmune conditions or trigger latent infections, making this period especially vulnerable to immune-related complications.
    Conclusion: Hormonal and immune responses are closely interdependent in the postpartum period, leading to heightened susceptibility to infections, autoimmune flare-ups, and other immune-related disorders. For improved postpartum care and enhanced maternal health outcomes, more research is necessary to clarify the mechanism of immune reconstitution, find possible hormonal indicators, and create focused therapeutic approaches. This review further highlights the critical role of hormonal-immune crosstalk in postpartum mood disorders (PPD, postpartum anxiety [PPA], and postpartum psychosis [PP]), proposing integrated biomarkers for early intervention.
    Keywords:  hormonal changes; immune system; inflammatory reactions; mother recovery; postpartum
    DOI:  https://doi.org/10.3389/abp.2025.14241
  2. Curr Opin Immunol. 2025 Jun 20. pii: S0952-7915(25)00071-8. [Epub ahead of print]95 102595
      Healthy individuals exhibit sex-specific immune responses. In certain immune-related diseases, such as autoimmune diseases, there is a pronounced sex bias in incidence, severity, and other clinical features. These differences are influenced by immune-related genes encoded in the sex chromosomes, as well as by genes whose expression is regulated by sex hormones. Sex hormone receptors play key roles in gene expression regulation by inducing epigenetic modifications and chromatin structure changes, thereby influencing the properties of immune cells at multiple levels. In this review, we provide an overview of the pathways activated by sex hormones and their receptors, highlighting their interplay with the epigenetic machinery that shapes the properties of immune cells in both females and males. We then examine the implications of sex hormone-associated epigenetic dysregulation in autoimmune diseases. Finally, we discuss current and potential clinical implications to enhance understanding of pathophysiology and the prospects for biomarkers and therapy. This review offers valuable insights into the roles of sex hormones and the mechanisms governing immune function and pathogenesis.
    DOI:  https://doi.org/10.1016/j.coi.2025.102595
  3. Endocrine. 2025 Jun 21.
       PURPOSE: The aim of this paper is to review the scientific evidence for the link between autoimmune thyroid diseases (AITDs) and sex, and the plausible causes of the female prevalence in AITDs.
    METHODS: An extensive literature search of published articles was conducted using online search engines.
    RESULTS: AITDs are more frequent in the female sex. Numerous studies have focused on the differences between males and females in their presentation of AITD and their response to therapy, sometimes with conflicting results. Several factors are considered to be responsible for the higher incidence of AITDs in females, crucial among which is the relationship between sex hormones and the immune system. In addition to hormones, there is also evidence that sex chromosomes, epigenetic factors, foetal microchimerism, the microbiota and endocrine-disrupting chemicals (EDCs) play an important role in the predisposition of females to AITDs.
    CONCLUSION: The prevalence of AITDs in the female sex appears to be attributable to numerous factors, the most important being the influence of sex hormones on the immune system.
    Keywords:  Autoimmune thyroid diseases; Endocrine-disrupting chemicals; Foetal microchimerism; Genes; Microbiota; Sex hormones
    DOI:  https://doi.org/10.1007/s12020-025-04317-5
  4. Diseases. 2025 Jun 07. pii: 179. [Epub ahead of print]13(6):
      In recent years, gender medicine has emerged as a field of research analyzing sex-related differences in health and disease. Biological sex, depending on sex chromosome complement, sex steroid hormones, and reproductive organs, has been demonstrated to influence human susceptibility to infections, immune responses against pathogens, the clinical severity of infectious diseases, and responses to the available treatments. Men and women differ in their chromosome set, with men having one X chromosome (XY) and women two (XX). This different genetic composition results in a sex-dimorphic expression of genes and pathways involved in immune regulation, as well as in shaping immune responses to infectious agents. Moreover, estrogen, progesterone, and testosterone, impacting cells and pathways involved in both innate and adaptive immunity, have been shown to drive sex dimorphism in infectious diseases. This narrative review aims to explore the sex-related differences in responses to infections, specifically focusing on the underlying genetic and hormonal mechanisms. Hence, aging-related changes in the immune system and their potential impact on immune responses against pathogens will be discussed. Understanding sex differences and stratifying the population according to them will open the door to precision medicine and personalized patient care.
    Keywords:  gender medicine; genetics; hormones; immune system; infectious diseases
    DOI:  https://doi.org/10.3390/diseases13060179
  5. Biomedicines. 2025 Jun 18. pii: 1495. [Epub ahead of print]13(6):
      Hashimoto's thyroiditis is the most prevalent autoimmune thyroid disorder, and it disproportionately affects women of reproductive age. Its impact on fertility and assisted reproductive technologies [ART] has become an area of growing clinical interest. Thyroid autoimmunity can influence female reproductive health through multiple interconnected mechanisms, including subtle thyroid hormone imbalances, reduced ovarian reserve, altered endometrial receptivity, and dysregulated immune responses. Subclinical hypothyroidism and the presence of anti-thyroid antibodies have been linked to increased miscarriage risk and reduced success rates in ART, particularly in intracytoplasmic sperm injection (ICSI) cycles. Although levothyroxine supplementation is widely used, its benefits in euthyroid women remain uncertain. Recent studies suggest that gut microbiota may modulate immune function and affect fertility outcomes among women with autoimmune thyroid conditions. This narrative review synthesizes findings from a broad literature base of over 40 peer-reviewed publications published between 2010 and 2025, with 30 of the most relevant and methodologically robust studies selected for detailed analysis. The review integrates clinical, endocrine, immunological, and microbiome-related perspectives. The evidence supports the need for personalized fertility management in women with Hashimoto's thyroiditis and highlights directions for future research into immune and microbiota-targeted therapies.
    Keywords:  immune dysregulation; ovarian reserve; thyroid autoantibodies
    DOI:  https://doi.org/10.3390/biomedicines13061495
  6. Semin Ophthalmol. 2025 Jun 24. 1-10
      Relationship between sex hormones and ocular surface immunity is controversial. Estrogens, androgens, and progesterone, play pivotal roles in modulating immune responses on the ocular surface. Estrogens exhibit dual roles, acting as both pro-inflammatory and anti-inflammatory agents depending on their concentration and receptor interaction, while androgens generally demonstrate immunosuppressive effects. Progesterone, though less studied, may have immunomodulatory properties. Despite numerous studies, the exact molecular pathways through which sex hormones regulate ocular immunity are not fully understood, and findings often appear inconsistent. To address these gaps, a comprehensive literature search was conducted across PubMed, Scopus, and Web of Science, focusing on keywords related to sex hormones, immunity, and ocular surface health. The aim is to summarize hormone receptor mechanisms, their influence on immune cell function and production of inflammatory mediators at the ocular surface. Overall, this review highlights the intricate interplay between sex hormones and ocular surface immunity, emphasizing that hormonal balance is essential for maintaining ocular health. A deeper understanding of these mechanisms could drive the development of novel, hormone-based therapeutic strategies for inflammatory ocular disorders, such dry eye disease, meeting an urgent clinical need.
    Keywords:  Androgen; dry eye; estrogen; ocular surface; progesterone; sex hormones
    DOI:  https://doi.org/10.1080/08820538.2025.2522716
  7. Life (Basel). 2025 Jun 05. pii: 916. [Epub ahead of print]15(6):
      The study of the microbiome has rapidly progressed over the past few decades, capturing the interest of both scientists and the general public. Nevertheless, there is still no widely agreed-upon definition for the term "microbiome" despite tremendous advances in our knowledge. The international scientific literature consistently underscores the difference between the human microbiome and human microbiota. Recent research has emphasized the importance of the female reproductive tract microbiome in fertility, impacting natural conception and assisted reproductive technologies (ARTs). This review explores the relationship between infertility and the microbiota of the female reproductive tract through a thorough evaluation of research papers and large-scale studies published up to 2024. The objective of this review is to critically assess current evidence on the role of the reproductive tract microbiome in female infertility and ART outcomes. Relevant papers were identified and analyzed through the electronic medical databases PubMed/MEDLINE and Scopus. A comprehensive synthesis of data from 36 original studies was performed, including observational, case-control, cohort, and randomized trials. By focusing on the vagina, cervix, and endometrium, this study offers a comprehensive overview of the microbiome throughout the female reproductive tract. RIF and poor reproductive outcomes are strongly linked to dysbiosis, which is characterized by a reduction in Lactobacillus species. Lactobacillus crispatus, in particular, plays a significant role in protecting against bacterial vaginosis and infertility. A thorough understanding of how the microbiome impacts fertility and the development of clinical strategies to improve reproductive outcomes requires standardized microbiome investigation techniques and larger, randomized trials that account for diverse patient characteristics.
    Keywords:  Lactobacillus; female infertility; female reproductive system; microbiome; microbiota
    DOI:  https://doi.org/10.3390/life15060916
  8. Ultrasound Obstet Gynecol. 2025 Jun 26.
       OBJECTIVES: A short cervix is a known risk factor for preterm birth, and imbalances in the vaginal microbiome, such as low relative abundance of Lactobacillus, may be associated with an increased risk of preterm birth. The aim of this study was to evaluate differences in the vaginal microbiome between women with a short cervix and those with normal cervical length in the second trimester. Additionally, we aimed to assess longitudinal changes in microbial diversity during pregnancy, as well as the impact of vaginal progesterone treatment on vaginal microenvironment in women with a short cervix.
    METHODS: This was a prospective, longitudinal study conducted at Koc University Hospital between January 2020 and May 2023, in women with a singleton pregnancy with a short cervical length (≤ 25 mm) in the second trimester (20 + 0 to 24 + 6 weeks' gestation). After diagnosis of short cervix, administration of 200 mg vaginal progesterone daily was initiated. The control group comprised women with a normal cervical length (> 25 mm) in the second trimester, matched for age and body mass index (BMI). Cervicovaginal swabs were collected from the posterior fornix at three gestational-age ranges: in the first trimester (11 + 0 to 13 + 6 weeks), the second trimester (20 + 0 to 24 + 6 weeks) and the third trimester (28 + 0 to 34 + 6 weeks), and cervical length was measured following sample collection. DNA was extracted and the 16S rRNA bacterial gene was sequenced to analyze and compare the vaginal microbiome between women with a short cervix and controls. We also assessed the microbiome longitudinally in each group, across the first, second and third trimesters. In the short-cervix group, we also compared the microbiome before initiation of progesterone treatment in the second trimester and 4 weeks after its initiation.
    RESULTS: Among 490 pregnant women who underwent first-trimester screening during the study period and had vaginal swabs collected, short cervical length was detected in 31 at the second-trimester scan. These women formed the study group. A further 27 women, with a normal cervical length, were matched for BMI and age and assigned to the control group. During the second trimester, women with a short cervix exhibited greater species diversity compared with the control group; this was suggested by the higher Shannon index (0.45 vs 0.33; P = 0.135), which reflects species richness and evenness, and further demonstrated by the higher Chao index (20.2 vs 13.8; P = 0.018), which estimates species richness. In the second trimester, Lactobacillus was less abundant in women with a short cervix than in the control group, although the difference did not reach significance (86.8% vs 95.5%; P = 0.091). At the phylum level, in women with a short cervix compared to those with normal cervical length, the relative abundance of Firmicutes, to which the genus Lactobacillus belongs, was significantly lower (90.7% vs 97.6%; P = 0.041), while the relative abundances of both Bacteroidota (1.73% vs 0.4%; P = 0.004) and Proteobacteria (0.2% vs 0.01%; P = 0.007) were higher. In the second trimester, the relative abundance of Lactobacillus gasseri was significantly lower in women with a short cervix compared to controls (4.7% vs 13.8%; P = 0.023). In the longitudinal analysis of the vaginal microbiome, there were no significant differences among the trimesters in the control group. In contrast, in those with a short cervix, there was a notable decrease in the amount of Lactobacillus crispatus, from 55.0% in the first trimester to 36.1% in the second trimester (P = 0.052). In women with a short cervix, there was no significant difference in bacterial diversity after vs before progesterone treatment (Chao index, 22.6 vs 20.5; P = 0.609).
    CONCLUSION: These findings highlight the significant alterations in the vaginal microbiome of pregnant women with a short cervix in comparison to those with normal cervical length, particularly in terms of higher species diversity and distinct community composition. The study also shows that vaginal progesterone treatment in women with a short cervix does not alter the vaginal microbiome, suggesting that it is a safe and effective intervention without disrupting the vaginal microbial balance. Understanding the relationship between cervical length and the vaginal microbiome is essential for developing strategies to reduce the risk of preterm birth in high-risk populations. © 2025 The Author(s). Ultrasound in Obstetrics & Gynecology published by John Wiley & Sons Ltd on behalf of International Society of Ultrasound in Obstetrics and Gynecology.
    Keywords:  cervical length; pregnancy; preterm birth; progesterone; vaginal microbiome
    DOI:  https://doi.org/10.1002/uog.29269
  9. Biomedicines. 2025 May 29. pii: 1332. [Epub ahead of print]13(6):
      The female genital microbiota plays a critical role in reproductive health and has recently emerged as a key factor influencing the outcomes of Assisted Reproductive Techniques (ARTs). Beyond traditional concerns about vaginal dysbiosis and infections such as bacterial vaginosis or mycoses, recent evidence highlights the broader impact of genital microbial communities, including the vaginal, cervical, and endometrial niches, on ART success rates. New findings suggest that specific bacterial profiles, as well as shifts in the virome and mycobiome, can significantly affect implantation and pregnancy outcomes. Non-invasive biomarkers such as menstrual blood have also been proposed for assessing endometrial receptivity. Furthermore, growing attention has been directed towards methodological challenges such as contamination risks during microbiota sampling which may influence study reliability. This review synthesizes the latest data on the relationship between the female genital microbiota and ART outcomes, with a focus on standardized microbiological analysis techniques and specific patient populations such as those experiencing recurrent implantation to optimize ART success based on microbiota profiling.
    Keywords:  ART; assisted reproductive techniques; bacteria; endometrial microbiota; fertility; genital microbiota; vaginal microbiota
    DOI:  https://doi.org/10.3390/biomedicines13061332
  10. Biomedicines. 2025 May 27. pii: 1314. [Epub ahead of print]13(6):
      Obesity is a major contributor to male infertility, not only exacerbating infertility but also impairing the effectiveness of both surgical interventions and medical treatments. This review examines the complex relationship between obesity, the immune microenvironment, and male infertility, highlighting how obesity-induced changes in immune function lead to testicular dysfunction and impaired spermatogenesis. Key mechanisms include chronic low-grade inflammation, immune cell infiltration, and dysregulated adipokines such as leptin and adiponectin. We also explore current therapeutic strategies aimed at alleviating these effects, including lifestyle interventions, anti-inflammatory treatments, metabolic therapies, and regenerative medicine approaches, such as exosome-based therapies. Despite promising results, substantial research gaps remain, particularly in understanding the molecular mechanisms and identifying novel biomarkers for early diagnosis. Future studies should focus on multi-omics approaches, large-scale cohort studies, the gut-testis axis, and the psychological and social factors influencing male infertility. A deeper understanding of these processes is crucial for developing more effective, targeted therapies for obesity-related male infertility.
    Keywords:  adipokines; blood–testis barrier; immune microenvironment; inflammatory cytokines; male infertility; obesity; oxidative stress; sperm quality; testicular dysfunction
    DOI:  https://doi.org/10.3390/biomedicines13061314
  11. Metabolites. 2025 Jun 11. pii: 390. [Epub ahead of print]15(6):
      The human gut microbiome is integral to maintaining systemic physiological balance, with accumulating evidence emphasizing its critical role in reproductive health. This review investigates the bidirectional interactions between the gut microbiota and the female reproductive system, mediated by neuroendocrine, immune, and metabolic pathways, constituting the gut-reproductive axis. Dysbiosis, characterized by microbial imbalance, has been linked to reproductive disorders such as polycystic ovary syndrome (PCOS), endometriosis, infertility, impaired spermatogenesis, and pregnancy complications. These associations can be explained by immunological dysregulation, systemic inflammation, altered sex hormone metabolism, and hypothalamic-pituitary-gonadal (HPG) axis disturbances. This review aims to clarify the molecular and cellular mechanisms underpinning gut-reproductive interactions and to evaluate the feasibility of microbiome-targeted therapies as clinical interventions for improving reproductive outcomes.
    Keywords:  fertility; gut microbiota; hormone; immune system; inflammation; microbiota–gonadal axis
    DOI:  https://doi.org/10.3390/metabo15060390
  12. Eur J Immunol. 2025 Jun;55(6): e51673
      The recent COVID-19 pandemic has highlighted a significant sex bias in disease outcome, where male sex is associated with greater disease severity and mortality. Interestingly, studies have also identified a role for antigen-independent "bystander-activated" CD8+ T cells in the severity of COVID-19 and other viral infections. However, whether biological sex contributes to the magnitude of bystander T cell activation has not been investigated. To assess sex differences in bystander CD8+ T cell activation, we isolated PBMCs from age-matched male and female donors and stimulated the cells with cytokines IL-12/15/18 to induce bystander T cell activation. Male CD8+ T cells stimulated with IL-15 exhibited greater bystander activation, including increased NKG2D expression and greater antigen-independent cytotoxicity against tumor cells compared with female CD8+ T cells. In contrast, IL-12/18 and IL-12/15/18 stimulation of CD8+ T cells did not reveal evidence of sex differences in bystander IFN-γ production. Our data suggest that underlying sex differences in bystander CD8+ T cell activation and cytotoxicity may contribute to the observed sex biases in disease severity of viral infections.
    Keywords:  CD8+ T cells; NKG2D; biological sex; bystander activation
    DOI:  https://doi.org/10.1002/eji.202451673
  13. Steroids. 2025 Jun 21. pii: S0039-128X(25)00096-0. [Epub ahead of print] 109655
       PURPOSE: This study aimed to evaluate the potential impact of serum E2 levels on vaginal pH regulation and Candida colonization in women undergoing infertility treatment, emphasizing mucosal barrier dynamics and epithelial immune modulation.
    METHODS: In this prospective observational study, 60 women aged 20-49 undergoing long-protocol ovulation induction were enrolled. Serum E2 levels, vaginal pH, and fungal cultures were assessed on cycle day 2 (baseline) and again at the time of estradiol peak, which was typically within 24 h before human chorionic gonadotropin (hCG) administration and after ultrasound-confirmed follicular maturation. Pre- and post-treatment paired samples were statistically compared, with significance set at p < 0.05.
    RESULTS: Post-treatment analysis revealed a significant increase in serum E2 and vaginal pH levels (p < 0.05). However, no significant correlation was observed between E2 and vaginal pH, Candida colonization, or vulvovaginal symptoms. Likewise, vaginal pH was not a significant predictor of fungal growth. Notably, a significant association between baseline symptoms and culture positivity was observed pre-treatment, which was not present post-treatment.
    CONCLUSION: These findings indicate that the hormonal increase in E2 during infertility treatment, although associated with elevated vaginal pH, does not increase the risk of Candida infections. The preservation of epithelial barrier function despite hormonal shifts may explain this finding. These results support the microbiological safety of controlled ovarian stimulation protocols with regard to Candida susceptibility.
    Keywords:  Candida albicans; Estradiol; Female; Infertility; Ovulation Induction
    DOI:  https://doi.org/10.1016/j.steroids.2025.109655
  14. Int J Reprod Biomed. 2024 Apr;23(3): 241-250
       Background: Women with low ovarian reserve are the capable part of people who refer to infertility centers, this study compares 2 treatment protocols in cases with low ovarian reserve.
    Objective: This study aimed to compare the efficacy of gonadotropin-releasing hormone (GnRH) agonist and GnRH-antagonist protocols in women with diminished ovarian reserve (DOR) in POSEIDON groups 3-4 undergoing in assisted reproductive technology (ART).
    Materials and Methods: This randomized clinical trial enrolled 158 infertile women with diminished ovarian reserve undergoing ART at the Research and Clinical Center for Infertility, Yazd, Iran from January to October 2024. Women were randomly assigned to either a GnRH-antagonist (n = 84) or a long GnRH-agonist (n = 74). Primary outcomes included clinical pregnancy and secondary outcomes were chemical pregnancy, early abortion rate, ongoing pregnancy, and implantation rate.
    Results: No significant differences were observed in baseline values between groups. The GnRH-agonist group had a longer stimulation duration (12.23 vs. 10.60 days, p < 0.001) and a higher total gonadotropin dose (4588.84 vs. 3225.67 IU, p < 0.001) compared to the antagonist group. Clinical pregnancy rates (17.8% vs. 15.8%, p = 0.810) and live birth rates (11.1% vs. 13.2%, p = 0.775) were comparable between the agonist and antagonist groups.
    Conclusion: According to acquired data, the GnRH-antagonist and long GnRH-agonist protocols resulted in similar ART outcomes. The antagonist protocol was associated with shorter stimulation and lower gonadotropin consumption. As a result, the antagonist protocol was found to be more cost effective. Larger studies are needed to confirm these results and determine the optimal protocol for this woman group.
    Keywords:   ART.; In vitro fertilization; Ovarian stimulation; Ovarian reserve
    DOI:  https://doi.org/10.18502/ijrm.v23i3.18775
  15. Drug Des Devel Ther. 2025 ;19 5255-5270
      Polycystic ovary syndrome (PCOS) is a common endocrine-metabolic disorder in women of reproductive age, marked by hyperandrogenism, ovulatory dysfunction, and insulin resistance, accompanied by significant metabolic disturbances, including glycolytic dysfunction, mitochondrial impairment, and increased oxidative stress. In granulosa cells (GCs), disrupted glycolysis impairs follicular development and compromises oocyte quality, exacerbating reproductive and metabolic abnormalities. At the molecular level, dysregulated energy-sensing pathways, such as AMPK and mTOR, reduce glucose uptake, lower ATP generation, and enhance oxidative stress, fueling disease progression. Epigenetic changes and non-coding RNAs further modulate glycolytic enzyme expression, destabilizing metabolic homeostasis within ovarian follicles. Therapeutically, restoring glycolytic balance using agents like metformin, resveratrol, mogroside V, and nicotinamide mononucleotide (NMN) has shown promise in improving glycolysis, insulin sensitivity, and ovarian function in various models. This review synthesizes current evidence on glycolysis's critical role in PCOS pathophysiology, its influence on follicular energetics and oocyte quality, and highlights metabolic targets for future therapies, offering a foundation for novel mechanism-driven interventions in PCOS management.
    Keywords:  glycolysis; granulosa cells; insulin resistance; metabolic dysfunction; metabolic therapy; oxidative stress; polycystic ovary syndrome
    DOI:  https://doi.org/10.2147/DDDT.S525651
  16. ISME J. 2025 Jun 27. pii: wraf055. [Epub ahead of print]
      Bacterial vaginosis (BV) is a common, polymicrobial condition of the vaginal microbiota that is associated with symptoms such as malodor and excessive discharge, along with increased risk of various adverse sequelae. Host-bacteria and bacteria-bacteria interactions are thought to contribute to the condition, but many of these functions have yet to be elucidated. Using untargeted metaproteomics, we identified 1068 host and 1418 bacterial proteins in a set of cervicovaginal lavage samples collected from 20 participants with BV and 9 who were negative for the condition. We identified Dialister micraerophilus as a major producer of malodorous polyamines and identified a syntrophic interaction between this organism and Fannyhessea vaginae that leads to increased production of putrescine, a metabolite characteristic of BV. Although formate synthesis has not previously been noted in BV, we discovered diverse bacteria associated with the condition express pyruvate formate-lyase enzymes in vivo and confirm these organisms secrete formic acid in vitro. Sodium hypophosphite efficiently inhibited this function in multiple taxa. We also found that the fastidious organism Coriobacteriales bacterium DNF00809 can metabolize formic acid secreted by Gardnerella vaginalis, representing another syntrophic interaction. We noted an increased abundance of the host epithelial repair protein transglutaminase 3 in the metaproteomic data, which we confirmed by enzyme-linked immunosorbent assay. Other proteins identified in our samples implicate Finegoldia magna and Parvimonas micra in the production of malodorous trimethylamine. Some bacterial proteins identified represent novel targets for future therapeutics to disrupt BV communities and promote vaginal colonization by commensal lactobacilli.
    Keywords:  bacterial vaginosis; drug targets; fermentation; host–microbe interactions; metabolism; metaproteomics; microbial ecology; microbiome; syntrophy
    DOI:  https://doi.org/10.1093/ismejo/wraf055