bims-raghud Biomed News
on RagGTPases in human diseases
Issue of 2024‒05‒12
four papers selected by
Irene Sambri, TIGEM



  1. Physiol Res. 2024 Apr 30. 73(2): 173-187
      Sodium is the main osmotically active ion in the extracellular fluid and its concentration goes hand in hand with fluid volume. Under physiological conditions, homeostasis of sodium and thus amount of fluid is regulated by neural and humoral interconnection of body tissues and organs. Both heart and kidneys are crucial in maintaining volume status. Proper kidney function is necessary to excrete regulated amount of water and solutes and adequate heart function is inevitable to sustain renal perfusion pressure, oxygen supply etc. As these organs are bidirectionally interconnected, injury of one leads to dysfunction of another. This condition is known as cardiorenal syndrome. It is divided into five subtypes regarding timeframe and pathophysiology of the onset. Hemodynamic effects include congestion, decreased cardiac output, but also production of natriuretic peptides. Renal congestion and hypoperfusion leads to kidney injury and maladaptive activation of renin-angiotensin-aldosterone system and sympathetic nervous system. In cardiorenal syndromes sodium and water excretion is impaired leading to volume overload and far-reaching negative consequences, including higher morbidity and mortality of these patients. Keywords: Cardiorenal syndrome, Renocardiac syndrome, Volume overload, Sodium retention.
  2. Semin Nephrol. 2024 May 06. pii: S0270-9295(24)00031-7. [Epub ahead of print] 151511
      
    DOI:  https://doi.org/10.1016/j.semnephrol.2024.151511
  3. JACC Heart Fail. 2024 May;pii: S2213-1779(24)00253-1. [Epub ahead of print]12(5): 860-863
      
    Keywords:  GFR decline; cardiokidney-metabolic syndrome; chronic kidney disease; heart failure
    DOI:  https://doi.org/10.1016/j.jchf.2024.02.028
  4. J Nephrol. 2024 May 04.
      The pathological features of acute and chronic kidney diseases are closely associated with cell death in glomeruli and tubules. Ferroptosis is a form of programmed cell death characterized by iron overload-induced oxidative stress. Ferroptosis has recently gained increasing attention as a pathogenic mechanism of kidney damage. Specifically, the ferroptosis signaling pathway has been found to be involved in the pathological process of acute and chronic kidney injury, potentially contributing to the development of both acute and chronic kidney diseases. This paper aims to elucidate the underlying mechanisms of ferroptosis and its role in the pathogenesis of kidney disease, highlighting its significance and proposing novel directions for its treatment.
    Keywords:  Ferroptosis; Iron; Kidney disease; Lipid peroxidation; Oxidative stress
    DOI:  https://doi.org/10.1007/s40620-024-01927-6