bims-ovdlit Biomed News
on Ovarian cancer: early diagnosis, liquid biopsy and therapy
Issue of 2026–07–26
eight papers selected by
Lara Paracchini, Humanitas Research



  1. Pathol Int. 2026 Jul;76(7): e70152
      Clusters of epithelial cells of the fallopian tube exhibiting aberrant p53 expression, termed "p53 signatures," are detected at a relatively high frequency and have been regarded as putative precursors of high-grade serous carcinoma (HGSC). In contrast, the recently proposed "β-catenin signature" represents a distinct tubal epithelial alteration characterized by the aberrant expression of β-catenin, the biological significance of which remains unclear. To clarify the frequency and spatial relationship between the two lesions, we analyzed 30 consecutive risk-reducing salpingo-oophorectomy specimens from BRCA1/2 mutation carriers using the sectioning and extensively examining the fimbriated end protocol and performed immunohistochemical evaluation for p53, β-catenin, and Ki-67. p53 signatures and β-catenin signatures were each identified in seven cases (23%). Two cases (6.7%) harbored both lesions; however, no spatial overlap or histological continuity was observed. Neither lesion demonstrated significant associations with BRCA mutation subtype, age, body mass index, or parity. Furthermore, no cases showed serous tubal intraepithelial lesion, serous tubal intraepithelial carcinoma, or HGSC. These findings suggest that p53 signatures and β-catenin signatures may represent distinct localized epithelial alterations in the fallopian tube, although further molecular and spatial analyses are required to clarify their biological relationship.
    Keywords:  BRCA mutation; fallopian tube; p53; β‐catenin
    DOI:  https://doi.org/10.1111/pin.70152
  2. Oncol Rev. 2026 ;20 1885622
      Head and neck cancer (HNC) diagnosis and surveillance still rely primarily on clinical examination, imaging, and tissue biopsy, with no established non-invasive biomarkers for routine early detection or disease monitoring. This limitation underscores the need for alternative strategies that can facilitate timely diagnosis and longitudinal assessment. Salivary cell-free DNA (scfDNA) has emerged as a promising liquid biopsy biomarker owing to its close proximity to primary HNC lesions and its suitability for non-invasive, repeatable sampling. Detection and characterization of scfDNA enable the identification of tumour-associated molecular alterations, including somatic mutations, aberrant DNA methylation, copy number alterations, viral DNA, and fragmentation signatures, providing valuable insights into tumour biology and disease dynamics. These molecular features have demonstrated considerable potential for early detection, prognostication, therapeutic monitoring, and post-treatment surveillance in head and neck cancer. However, current data are derived largely from exploratory studies with heterogeneous methodologies, small validation cohorts, and limited standardization of analytical workflows. Nevertheless, advances in sample processing, extraction methods, high-sensitivity sequencing technologies, and advanced bioinformatic approaches are enhancing assay accuracy and reproducibility. This review summarizes the current evidence on scfDNA in HNC by delineating its biological basis, evaluating the robustness and translational relevance of available clinical studies, and examining the analytical, technical, and implementation challenges that must be addressed to facilitate its integration into evidence-based clinical practice.
    Keywords:  diagnosis; head and neck cancer; liquid biopsy; minimal residual disease; prognosis; salivary cell-free DNA; therapeutic monitoring
    DOI:  https://doi.org/10.3389/or.2026.1885622
  3. Nat Rev Clin Oncol. 2026 Jul 24.
      Analysis of tissue biopsy samples is the gold-standard approach to cancer diagnosis and patient selection for biomarker-guided therapies. Although spatial analyses of tumour tissue can provide important insights into local antitumour immune responses, repeated tumour biopsy is invasive and rarely feasible for monitoring dynamic immune responses over time. Technical advances have enabled multimodal analyses of cells and cellular products in peripheral blood samples, which can be obtained easily and repeatedly over the course of the disease. These liquid biopsy-based approaches cannot provide spatial information on the tumour immune microenvironment and tumour-derived material can be highly diluted in the circulation, yet they offer a unique opportunity to monitor systemic immune dynamics, anticipate responses to immunotherapies and detect emerging resistance to treatment. Thus, blood-based analyses might complement tumour tissue analysis. In this Review, we discuss insights on systemic antitumour immune responses that can be gained through the analysis of tumour-derived biomarkers in peripheral blood and/or circulating immune cells.
    DOI:  https://doi.org/10.1038/s41571-026-01181-8
  4. J Gastrointest Cancer. 2026 Jul 21. pii: 160. [Epub ahead of print]57(1):
      Gastroesophageal cancers (GEC) have high mortality rates with poor prognosis, especially at late stages. This is partly due to unspecific symptoms at early stages, early treatment resistance, and recurrence after curative-intent treatment. Circulating tumor DNA (ctDNA) has gained attention for early cancer diagnosis and longitudinal monitoring. This narrative review summarizes the recent developments of genomic ctDNA analysis for early GEC diagnosis and longitudinal monitoring across 57 relevant studies. For early diagnosis, several studies combined genomic ctDNA analysis with other ctDNA approaches or serum biomarkers. These approaches achieved high sensitivities and specificities in cancer detection within GEC while accurately predicting the tissue of origin, The use of genomic ctDNA analysis has also been studied for longitudinal monitoring of GEC patients during and after various treatment regimens. Studies have shown that ctDNA dynamics is highly consistent with treatment response and disease progression. CtDNA could frequently detect disease progression before conventional methods, thereby displaying its potential application in longitudinal monitoring. Genomic ctDNA analysis could also detect recurrence after curative-intent treatment and, in many cases, before clinical detection. However, ctDNA has low abundance in total cell-free DNA during low-burden disease. Additionally, methodological variation in ctDNA processing and quantification complicates interpretation across studies.
    Keywords:  Biomarker; Circulating tumor DNA; Early diagnosis; Esophageal cancer; Gastric cancer; Longitudinal monitoring
    DOI:  https://doi.org/10.1007/s12029-026-01530-x
  5. Mol Oncol. 2026 Jul 23.
      Cancer develops inside organized tissue environments wherein cellular behavior is heavily influenced by local interactions and spatially restricted regulatory programs. While bulk and single-cell sequencing technologies have fundamentally revolutionized our understanding of tumor biology, these techniques often disrupt tissue architecture and therefore fail to capture the spatial context in which molecular processes occur. Spatial transcriptomics has provided important insights into tumor heterogeneity, microenvironmental organization, and cell-to-cell communication. However, gene expression alone offers only an indirect view of the regulatory mechanisms governing cellular states. The emergence of spatial epigenomic technologies now enables the investigation of chromatin accessibility, histone modifications, and DNA methylation while preserving tissue structure. Here, we discuss the current landscape of spatial epigenomics, including spatial ATAC-seq, spatial CUT&Tag, emerging spatial CUT&RUN approaches, spatial DNA methylation profiling, and multimodal strategies integrating epigenetic, transcriptional, and proteomic information within the same tissue context. Despite remaining technical and computational challenges, continued advances are expected to establish spatial epigenomics as a powerful tool for studying cancer pathways and their regulation within intact tissues.
    Keywords:  DNA methylation; cancer; chromatin; epigenetics; histone modifications; spatial biology
    DOI:  https://doi.org/10.1002/1878-0261.70310