bims-orenst Biomed News
on Organs-on-chips and engineered stem cell models
Issue of 2022‒05‒08
five papers selected by
Joram Mooiweer
University of Groningen


  1. ACS Biomater Sci Eng. 2022 May 03.
      A comparatively straightforward approach to accomplish more physiological realism in organ-on-a-chip (OoC) models is through substrate geometry. There is increasing evidence that the strongly, microscale curved surfaces that epithelial or endothelial cells experience when lining small body lumens, such as the alveoli or blood vessels, impact their behavior. However, the most commonly used cell culture substrates for modeling of these human tissue barriers in OoCs, ion track-etched porous membranes, provide only flat surfaces. Here, we propose a more realistic culture environment for alveolar cells based on biomimetically microcurved track-etched membranes. They recreate the mainly spherical geometry of the cells' native microenvironment. In this feasibility study, the membranes were given the shape of hexagonally arrayed hemispherical microwells by an innovative combination of three-dimensional (3D) microfilm (thermo)forming and ion track technology. Integrated in microfluidic chips, they separated a top from a bottom cell culture chamber. The microcurved membranes were seeded by infusion with primary human alveolar epithelial cells. Despite the pronounced topology, the cells fully lined the alveoli-like microwell structures on the membranes' top side. The confluent curved epithelial cell monolayers could be cultured successfully at the air-liquid interface for 14 days. Similarly, the top and bottom sides of the microcurved membranes were seeded with cells from the Calu-3 lung epithelial cell line and human lung microvascular endothelial cells, respectively. Thereby, the latter lined the interalveolar septum-like interspace between the microwells in a network-type fashion, as in the natural counterpart. The coculture was maintained for 11 days. The presented 3D lung-on-a-chip model might set the stage for other (micro)anatomically inspired membrane-based OoCs in the future.
    Keywords:  alveolar epithelial cells; biomimetics; curvature; ion track-etched membranes; microthermoforming; organ on a chip (OoC)
    DOI:  https://doi.org/10.1021/acsbiomaterials.1c01463
  2. Mater Today Bio. 2022 Mar;14 100259
      Models of heart disease and drug responses are increasingly based on human pluripotent stem cells (hPSCs) since their ability to capture human heart (dys-)function is often better than animal models. Simple monolayer cultures of hPSC-derived cardiomyocytes, however, have shortcomings. Some of these can be overcome using more complex, multi cell-type models in 3D. Here we review modalities that address this, describe efforts to tailor readouts and sensors for monitoring tissue- and cell physiology (exogenously and in situ) and discuss perspectives for implementation in industry and academia.
    Keywords:  Cardiac microtissue; Cardiomyocyte maturation; Engineered heart tissue; Functional readout; Heart-on-chip; Human induced pluripotent stem cells; Multicellular cell diseases and drug efficacy platform; Structural readout
    DOI:  https://doi.org/10.1016/j.mtbio.2022.100259
  3. Sci Adv. 2022 May 06. 8(18): eabm8012
      Protozoan parasites that infect humans are widespread and lead to varied clinical manifestations, including life-threatening illnesses in immunocompromised individuals. Animal models have provided insight into innate immunity against parasitic infections; however, species-specific differences and complexity of innate immune responses make translation to humans challenging. Thus, there is a need for in vitro systems that can elucidate mechanisms of immune control and parasite dissemination. We have developed a human microphysiological system of intestinal tissue to evaluate parasite-immune-specific interactions during infection, which integrates primary intestinal epithelial cells and immune cells to investigate the role of innate immune cells during epithelial infection by the protozoan parasite, Toxoplasma gondii, which affects billions of people worldwide. Our data indicate that epithelial infection by parasites stimulates a broad range of effector functions in neutrophils and natural killer cell-mediated cytokine production that play immunomodulatory roles, demonstrating the potential of our system for advancing the study of human-parasite interactions.
    DOI:  https://doi.org/10.1126/sciadv.abm8012
  4. Adv Mater. 2022 May 06. e2202992
      Epithelial cell organoids have increased opportunities to probe questions on tissue development and disease in vitro and for therapeutic cell transplantation. Despite their potential, current protocols to grow these organoids almost exclusively depend on culture within three-dimensional (3D) Matrigel, which limits defined culture conditions, introduces animal components, and results in heterogenous organoids (i.e., shape, size, composition). Here, we describe a method that relies on polymeric hydrogel substrates for the generation and expansion of lung alveolar organoids (alveolospheres). Using synthetic hydrogels with defined chemical and physical properties, human induced pluripotent stem cell (iPSC)-derived alveolar type 2 cells (iAT2s) self-assemble into alveolospheres and propagate in Matrigel-free conditions. By engineering pre-defined microcavities within these hydrogels, the heterogeneity of alveolosphere size and structure is reduced when compared to 3D culture, while maintaining alveolar type 2 cell fate of human iAT2-derived progenitor cells. This hydrogel system is a facile and accessible culture system for the culture of iPSC-derived lung progenitors and the method can be expanded to the culture of primary mouse tissue-derived AT2 and other epithelial progenitor and stem cell aggregates. This article is protected by copyright. All rights reserved.
    Keywords:  biomaterials; hyaluronic acid; hydrogels; lung; organoids
    DOI:  https://doi.org/10.1002/adma.202202992
  5. Immunity. 2022 Apr 27. pii: S1074-7613(22)00176-5. [Epub ahead of print]
      Macrophage colony stimulating factor-1 (CSF-1) plays a critical role in maintaining myeloid lineage cells. However, congenital global deficiency of CSF-1 (Csf1op/op) causes severe musculoskeletal defects that may indirectly affect hematopoiesis. Indeed, we show here that osteolineage-derived Csf1 prevented developmental abnormalities but had no effect on monopoiesis in adulthood. However, ubiquitous deletion of Csf1 conditionally in adulthood decreased monocyte survival, differentiation, and migration, independent of its effects on bone development. Bone histology revealed that monocytes reside near sinusoidal endothelial cells (ECs) and leptin receptor (Lepr)-expressing perivascular mesenchymal stromal cells (MSCs). Targeted deletion of Csf1 from sinusoidal ECs selectively reduced Ly6C- monocytes, whereas combined depletion of Csf1 from ECs and MSCs further decreased Ly6Chi cells. Moreover, EC-derived CSF-1 facilitated recovery of Ly6C- monocytes and protected mice from weight loss following induction of polymicrobial sepsis. Thus, monocytes are supported by distinct cellular sources of CSF-1 within a perivascular BM niche.
    Keywords:  LEPR; LPS; Ly6Chigh monocytes; Ly6Cnegative monocytes; MCSF1; bone development; bone marrow niche; endothelial cell; hematopoeisis; lipopolysaccharide; macrophage; macrophage colony stimulating factor; mesenchymal stromal cell; monocyte; monopoiesis; osteoblast; osteocyte; osteopetrosis; perivascular niche; perivascular stromal cell; sepsis; sinusoid
    DOI:  https://doi.org/10.1016/j.immuni.2022.04.005