bims-fragic Biomed News
on Fragmentomics
Issue of 2026–03–29
three papers selected by
Laura Mannarino, Humanitas Research



  1. Bioinformatics. 2026 Mar 26. pii: btag152. [Epub ahead of print]
       SUMMARY: Liquid biopsy offers a non-invasive approach to study tumor-derived genetic material circulating in plasma. Beyond genetic alterations, the fragmentomic features of cell-free DNA-such as fragment size, genomic position, and end-motifs-provide valuable insights into the biological and clinical context of DNA release. fRagmentomics is a user-friendly R package designed to characterize cfDNA fragments overlapping one or multiple small mutations of any type, starting from an aligned sequencing file (BAM). It supports multiple mutation input formats, accommodates one-based and zero-based genomic conventions, resolves mutation representation ambiguities, and accepts any reference file in FASTA format. For each fragment overlapping a mutation of interest, fRagmentomics outputs fragment-level features including its fragment size, end-motifs, and mutational status, along with additional fragment-level or read-level information. The package implements an indel-aware and optionally soft-clip-preserving fragment size computation that improves accuracy over conventional size estimates based solely on aligned positions.
    AVAILABILITY AND IMPLEMENTATION: fRagmentomics is licensed under GNU General Public License v3.0 and available at https://github.com/ElsaB-Lab/fRagmentomics and https://anaconda.org/elsab-lab/r-fragmentomics, with documentation and a tutorial.
    SUPPLEMENTARY INFORMATION: Supplementary data are available at Bioinformatics online.
    DOI:  https://doi.org/10.1093/bioinformatics/btag152
  2. Cancer Res Commun. 2026 Mar 25.
      Genome-wide coverage patterns of plasma cell-free DNA (cfDNA) fragments reflect nucleosome positioning in the cells of origin, enabling non-invasive inference of cell-type contributions and transcriptional activity. While the majority of cfDNA originates from hematopoietic cells, the diagnostic and biological relevance of this fraction remains underexplored. Here, we performed cfDNA-based deconvolution of blood cell types by integrating transcription start site (TSS) coverage profiles from plasma whole-genome sequencing with single-cell transcriptomic reference data. By correlating cfDNA TSS coverage with gene expression across 457 blood cell types, we ranked their relative contributions to the cfDNA pool. We analyzed 788 pre-treatment and longitudinal plasma samples from patients with localized colorectal cancer (CRC), muscle-invasive bladder cancer (MIBC), as well as 30 samples from healthy controls. In healthy individuals, cfDNA TSS coverage profiles reflected blood gene expression, and the inferred cell type contributions recapitulated the known hematopoietic composition. In cancer patients, we observed a significant increase in cfDNA contributions from lymphocytes, including T cells and plasma cells, and decreased contributions from monocytes and granulocytes. These immune-derived signatures distinguished CRC (AUC=0.793) and MIBC (AUC=0.745) patients from healthy controls. Longitudinal analysis of immune cell-type contributions revealed treatment-associated changes in the relative abundance of classical monocytes and plasma cells, although these temporal dynamics were not predictive of relapse or outcome. Together, these findings suggest that cfDNA-derived immune signatures may capture aspects of systemic immune remodeling in cancer, potentially providing a complementary non-invasive biomarker in liquid biopsies beyond tumor-derived signals.
    DOI:  https://doi.org/10.1158/2767-9764.CRC-25-0747
  3. PLoS Comput Biol. 2026 Mar 24. 22(3): e1014076
      Epigenetic processes, particularly disruptions in DNA methylation profiles, are associated with many disease states. Traditional approaches for DNA methylation biomarker discovery focusing on individual CpG sites do not account for fragment-level methylation states. Methylation haplotype analysis offers a more comprehensive approach leading to increased distinction capability between reads originating from tissues with diverse methylation profiles. This can particularly be valuable in liquid biopsy where detecting small amounts of disease-specific cell-free DNA (cfDNA) amidst a bulk of healthy cfDNA is challenging. To address limitations of existing metrics for quantifying methylation patterns in a region, we propose the Methylation Pattern Consistency Index (MPCI), a novel metric that captures consistent methylation patterns across sequencing reads, accounting for both methylated and unmethylated blocks of CpGs. Using whole-genome bisulfite sequencing data, we demonstrate that MPCI outperforms MHL and its symmetric counterpart, dMHL (MHL - uMHL), across several benchmarks: distinguishing closely related cell types (CD4 vs. CD8; AUC 0.915), multi-tissue classification (0.92 accuracy), and detection of in-silico cfDNA spike-ins at abundances as low as 1%. Notably, in a clinical liquid-biopsy cohort of liver transplant patients, MPCI achieved significantly higher classification performance than dMHL (Accuracy: MPCI: 0.868 ± 0.023 vs. dMHL: 0.768 ± 0.027, p = 0.014) in discriminating pre- from post-transplant cfDNA profiles. These findings position MPCI as a reliable quantification approach for biomarker selection or diagnostic testing in epigenetic studies. We have made MPCI available as an R function for usage convenience.
    DOI:  https://doi.org/10.1371/journal.pcbi.1014076