bims-exocan Biomed News
on Exosomes roles in cancer
Issue of 2026–02–22
six papers selected by
Muhammad Rizwan, COMSATS University



  1. ChemMedChem. 2026 Feb 25. 21(4): e202500653
      Rare cancers collectively account for a proportion of cancer-related morbidity and mortality, and patients face significant challenges, including delayed diagnosis, lack of targeted therapies, and poor clinical outcomes. Exosome-based therapies have emerged as promising tools to address these unmet needs. Exosomes, naturally secreted extracellular vesicles, are increasingly engineered as nanocarriers for the targeted delivery of chemotherapeutics, nucleic acids, and immune modulators. Their ability to modulate the tumor microenvironment, influence immune responses, and overcome drug resistance makes them especially attractive. In rare cancers, preliminary studies have demonstrated the utility of exosomes in improving tumor specificity, enhancing payload stability, and reducing systemic toxicity. Moreover, exosomes derived from tumor or immune cells can influence immune evasion, angiogenesis, and stromal remodeling, key processes in cancer progression. Despite this potential, the clinical application of exosome-based therapies in rare cancers remains underexplored. This review critically evaluates the limited but emerging body of evidence supporting exosome-based interventions in rare malignancies. By highlighting their therapeutic promise, we aim to understand exosome-driven strategies as personalized, effective, and accessible solutions for patients with rare cancers.
    Keywords:  exosome engineering; exosome‐based therapeutics; rare cancers; targeted drug delivery; tumor microenvironment modulation
    DOI:  https://doi.org/10.1002/cmdc.202500653
  2. Front Mol Biosci. 2026 ;13 1669297
      Skin malignancies, including melanoma and non-melanoma cancers, are the most common cancers worldwide, with increasing incidence and fatality rates. Malignant melanoma (MM) is a highly aggressive cancer with poor prognosis, and despite various therapies, it remains a serious threat due to factors like tumor recurrence, drug resistance, and lack of effective treatments. Non-coding RNAs (ncRNAs) transcripts have gained attention due to their critical roles in regulating proliferation, angiogenesis, immune regulation, invasion, metastasis, and treatment resistance. Exosomes, biologically active lipid-bilayer extracellular vesicles secreted by various cell types, are also involved in cancer by carrying multiple bioactive molecules, including ncRNAs. Investigating the noncoding components of the transcriptome and their exosomal counterparts opens up the possibility of discovering new therapeutic and diagnostic targets. This review discusses current studies on the involvement of ncRNAs and their exosomal counterparts in the pathogenesis, diagnosis, and treatment of human skin cancers, particularly melanoma.
    Keywords:  diagnosis; exosomes; melanoma; non-coding RNAs; skin cancer; therapeutictargets
    DOI:  https://doi.org/10.3389/fmolb.2026.1669297
  3. Int Rev Cell Mol Biol. 2026 ;pii: S1937-6448(25)00006-1. [Epub ahead of print]399 43-112
      Cancer remains a major health challenge, with traditional monitoring approaches that are often limited in sensitivity and invasiveness. Liquid biopsies, particularly those involving circulating extracellular vesicles (EVs), have emerged as a promising non-invasive approach that holds efficient diagnostic and prognostic value, predicting disease development and patient outcomes. Furthermore, EVs can monitor therapy responses and detect potential resistance development. In this review, we delineate the diverse cargo within EVs, including proteins, lipids, RNAs, and DNA, and their potential implications for cancer progression. The clinical utilization of EVs as prognostic and treatment-monitoring biomarkers for various types of cancer is addressed in depth, with a focus on their ability to identify molecular signatures associated with drug resistance. We also present a comprehensive collection of studies on EV-derived biomarkers for monitoring treatment responses across various cancer types, as well as investigations into EV-derived biomarkers associated with therapeutic resistance. While the promise of EV biomarkers in cancer therapy monitoring is acknowledged, we also discuss in detail the existing challenges and future directions in this research area. This review ultimately improves our understanding of liquid biopsies, specifically EV biomarkers, paving the way for more personalized and effective dynamic biomarkers.
    Keywords:  Biomarkers; Drug resistance; Exosomes; Extracellular vesicles; Liquid biopsy; Treatment monitoring
    DOI:  https://doi.org/10.1016/bs.ircmb.2025.01.006
  4. Mol Biol Rep. 2026 Feb 19. 53(1): 400
      Glioblastoma (GBM) leverages exosomes to drive tumor progression, immunosuppression, and therapy resistance through transfer of oncogenic miRNAs, circRNAs, and proteins. These vesicles reprogram the tumor microenvironment (TME) by activating pathways such as TREM1 in microglia and STAT3-mediated vasculogenic mimicry. Exosomal nucleic acids, including circZNF800 and miR-374b-3p, serve as non-invasive biomarkers for recurrence and treatment monitoring. Advances in engineering, such as Angiopep-2-functionalized exosomes for blood-brain barrier penetration and CRISPR-Cas9-loaded vesicles targeting resistance genes, highlight their therapeutic potential. Challenges in heterogeneity, standardization, and scalable production underscore the need for interdisciplinary innovation to translate exosome-based strategies into clinical practice.
    Keywords:  Biomarkers; Exosomes; Glioblastoma; Therapeutic engineering; Tumor microenvironment
    DOI:  https://doi.org/10.1007/s11033-026-11526-2
  5. Discov Oncol. 2026 Feb 19.
      
    Keywords:  Biomarkers; Cancer progression; Exosomal circular RNAs; Signaling pathways; Therapeutic Targets; Tumor apoptosis
    DOI:  https://doi.org/10.1007/s12672-026-04687-w
  6. PLoS One. 2026 ;21(2): e0342052
      Cancer-associated cachexia is a multifactorial syndrome characterized by significant weight loss, primarily due to skeletal muscle atrophy. This condition impairs the quality of life and survival of patients with cancer. Although the mechanisms underlying cancer-associated cachexia, including exosomes and microRNAs (miRNAs), have been extensively explored, research specifically focusing on cancer-associated fibroblast (CAF)-derived exosomes is lacking. Therefore, in this study, we evaluated the effects of CAF-derived exosomal miRNAs from colon cancer on skeletal muscles using the Human Skeletal Muscle (HSkM) cell line. CAF-derived exosomes were isolated from colon cancer samples, and their effects on cell morphology were analyzed using confocal microscopy. The results indicate that treatment with CAF-derived exosomes significantly reduced myosin diameter. Moreover, miRNA sequencing revealed that miR-125b was enriched in CAF-derived exosomes. HSkM cells were subsequently transfected with a miR-125b mimic, which significantly reduced myosin diameter. Notably, co-treatment with CAF-derived exosomes and an miR-125b inhibitor reversed this effect. In conclusion, this study demonstrates the potential role of CAF-derived exosomes and miR-125b in cancer-associated cachexia, offering insights into the contribution of the tumor microenvironment and suggesting possible therapeutic targets.
    DOI:  https://doi.org/10.1371/journal.pone.0342052